The incubation period between infection and theĭevelopment of clinical signs can be very variable. Such is associated with a high prevalence of latent inapparent infection, in which goats

The development of any clinical disease after infection will be slow and protracted, and as Hence potentially many kids – and this is also true for Johne’s disease. Herds can also lead to increased infection rates as one infected doe can infect the pool, and The practice of feeding pooled colostrum and milk in dairy Increased infection rates will occur in housed and intensively managed Minimal risk of transferring infection, both practices need to be risk managed if eradication isīeing attempted. Levels, and although both embryo transfer and artificial insemination may pose only a Transplacental infection can occur but is thought to be at low Milking parlour, or through shared use of equipment such as drenching guns To nose contact and aerosol transfer, via “infected milk impacts” at the teat end in the

There are however many other important routes of infection such as via nose This knowledge is vitalĬAE is transmitted predominantly via the ingestion of infected colostrum or milk from How the infectious agent is picked up and spread within a population. Occur, and is an important factor in CAE control programmes, that have been developed onĪ compulsive / voluntary basis in many countries around the world.Īn important part of any disease control or preventative programme, is an understanding of Although the two viruses causeĭifferent clinical presentations in their respective host species, cross species infection can Referred to collectively as SLRV (small ruminant lentivirus). Insidious conditions such as CAE and Maedi Visna (MV) of sheep, the two diseases often “The Veterinary Guide to Goat Health and Welfare”Ī lentivirus in the family Retroviridae, a genus causing a group of slowly developing

Signs of cae in goats